Haloxyl

Haloxyl Kalpa Pharmaceuticals

Haloxyl by Kalpa Pharmaceuticals is an oral steroid with active subtance Fluoxymesterone.

Haloxyl has strong androgenic properties and will give user strength and if used towards end of cycle will work well towards better hardness effect.

Haloxyl (Fluoxymesterone) is an oral anabolic steroid derived from testosterone. More specifically, it is a methyltestosterone derivative, differing by the addition of 11-beta-hydroxy and 9-alpha-fluoro groups. The result is a potent orally active non-aromatizable steroid that exhibits extremely strong androgenic properties.

Fluoxymesterone is considerably more androgenic than testosterone, while at the same time the anabolic effects of this agent are considered to be moderate in comparison. This makes fluoxymesterone a great strength drug, but not the most ideal agent for gaining muscle mass. The predominant effects seen when taking fluoxymesterone are increased strength, increased muscle density, and increased definition, with only modest size increases.

Haloxyl (Fluoxymesterone) is a modified form of testosterone. It differs by 1) the addition of a methyl group at carbon 17-alpha, which helps protect the hormone during oral administration, 2) the introduction of a fluoro group at carbon 9 (alpha) and 3) the attachment of a hydroxyl group at carbon 11 (beta), which inhibits steroid aromatization. The latter two modifications also greatly enhance the androgenic and relative biological activity of the steroid over 17-alpha methyltestosterone.

Haloxyl steroid cycle

Since it affects liver high dosages and long cycles must be avoided. When I say long I mean 5 weeks and dosage not more than 40mg/day.

The Haloxyl (read Fluoxymesterone) is not for newbies and must be always started in small dosages like 10mg/day to see how it works for me.

As a test cycle it can be run for 2 weeks at 10-20mg/day.

Taking 10mg before workout will increase energy and power.

Half life is around 8 hours.

Do NOT stack with other orals.

Fluoxymesterone and natural testosterone production

Steroids, particularly anabolic-androgenic steroids (AAS), exert a profound impact on the body's natural testosterone production. Upon administration, these exogenous hormones mimic the actions of endogenous testosterone, leading to significant increases in muscle mass, strength, and performance enhancement. However, this exogenous influx can disrupt the hypothalamic-pituitary-gonadal (HPG) axis, the endocrine system responsible for the regulation and production of testosterone.

The negative feedback loop of the HPG axis is crucial for maintaining homeostasis in testosterone levels. When exogenous steroids are introduced, the body detects the elevated androgen levels and, in response, the hypothalamus reduces the secretion of gonadotropin-releasing hormone (GnRH), which in turn causes the pituitary gland to lower the release of luteinizing hormone (LH) and follicle-stimulating hormone (FSH). As a result, the Leydig cells in the testes receive less LH signaling, leading to a decrease in endogenous testosterone production. Additionally, the elevated androgen levels can inhibit the feedback to the hypothalamus and pituitary, potentially causing further suppression of the HPG axis.

PCT after haloxyl steroid cycle

Following a steroid cycle, it is essential to implement a Post Cycle Therapy (PCT) with Clomid (Clomiphene Citrate), to restore the natural testosterone production. Clomid acts by blocking estrogen receptors in the hypothalamus and pituitary gland, thereby reducing the negative feedback effect of estrogen on the HPG axis. This allows the hypothalamus to detect lower estrogen levels and resume the production of GnRH, which then stimulates the pituitary to produce LH and FSH, signaling the testes to start producing testosterone again.

Moreover, PCT with Clomid may also help in preventing the loss of muscle mass and strength that typically occurs after a cycle due to the sudden drop in androgen levels, a condition known as post-cycle crash or hypogonadism. The use of Clomid may be complemented with other products such as human chorionic gonadotropin (hCG), which directly stimulates the Leydig cells to produce testosterone, and/or selective estrogen receptor modulators (SERMs) like Nolvadex (Tamoxifen).

Blood work is an essential component of monitoring the endocrine system's response to steroid use and PCT and after PCT. It allows for the measurement of various hormonal markers, including testosterone, estrogen, and luteinizing hormone, to assess the degree of HPG axis suppression and the efficacy of the PCT in re-establishing normal endogenous hormone production. Regular blood tests can help determine if adjustments are needed in the PCT protocol and provide insight into the individual's overall hormonal health, ensuring the timely detection and management of any potential adverse effects or complications.

In conclusion, steroids can significantly suppress natural testosterone production through negative feedback on the HPG axis. To mitigate this suppression and facilitate the body's return to homeostasis, a structured PCT with Clomid (and possibly other agents) is recommended. Blood work is a critical tool in monitoring the hormonal changes during this period to ensure the safety and effectiveness of the therapy. The scientific rationale behind PCT lies in the restoration of endogenous hormone levels and the minimization of the risks associated with steroid-induced hormonal imbalances.

Dont ignore PCT!